ISSN 1671-3710
CN 11-4766/R
主办:中国科学院心理研究所
出版:科学出版社

心理科学进展 ›› 2026, Vol. 34 ›› Issue (7): 1219-1238.doi: 10.3724/SP.J.1042.2026.1219 cstr: 32111.14.2026.1219

• 研究前沿 • 上一篇    下一篇

母性焦虑驱动过度保护行为: 情绪-认知-行为闭环模型

滕跃, 孔令楠, 刘丽芬, 杨彬, 党琪, 高军   

  1. 西南大学心理学部; 西南大学认知与人格教育部重点实验室, 重庆 400715
  • 收稿日期:2025-11-23 出版日期:2026-07-15 发布日期:2026-05-11
  • 通讯作者: 高军, E-mail: gaojunscience@126.com
  • 基金资助:
    国家自然科学基金项目(32071059); 重庆市社科规划项目(2024YC046); 重庆市社科规划项目(2020PY64)

Maternal anxiety-driven overprotective behaviors: An emotional-cognitive-behavioral closed-loop model

TENG Yue, KONG Lingnan, LIU Lifen, YANG Bin, DANG Qi, GAO Jun   

  1. Faculty of Psychology, Southwest University; Key Laboratory of Cognition and Personality, Ministry of Education, Southwest University, Chongqing 400715, China
  • Received:2025-11-23 Online:2026-07-15 Published:2026-05-11

摘要: 母性焦虑在当代育儿情境中日益普遍, 但其驱动并维持过度保护行为的机制仍缺乏系统性解释。本文围绕“母性焦虑如何通过多层心理与神经过程形成过度保护行为”这一核心问题, 整合临床、发展与神经科学领域的证据, 提出了一个创新的“情绪-认知-行为闭环模型”。该模型指出, 母性焦虑首先在情绪层面表现为威胁放大和情绪敏感失衡(杏仁核过度激活), 继而在认知层面引发注意偏向、灾难化思维(前额叶调控不足), 最终在行为层面通过“焦虑缓解”的负强化机制固化过度保护模式(腹侧被盖区-伏隔核奖赏通路参与其中)。情绪反应、认知偏差与行为强化相互作用, 构成一个自我维持的心理-神经闭环系统。该模型不仅整合了分散的研究证据, 也为未来的纵向研究、跨文化验证以及心理-神经多层干预(如认知重评、亲子互动训练与神经调控技术)提供了系统化理论框架。

关键词: 母性焦虑, 过度保护, 情绪-认知-行为闭环, 神经机制

Abstract: Maternal anxiety has become increasingly prevalent in contemporary parenting contexts; however, the mechanisms through which it drives and maintains overprotective behavior still lack a systematic theoretical explanation. To address this gap, the present review adopts a clinical perspective on maternal anxiety and synthesizes evidence from clinical, developmental, and neuroscientific research to propose an innovative “emotional-cognitive-behavioral closed-loop model” explaining the mechanisms underlying overprotective behavior. This model not only deepens our understanding of the relationship between maternal anxiety and parenting behavior but also offers new directions for theoretical innovation in integrated psychological, neural, and social interventions.
The model proposes that maternal anxiety drives overprotective behavior through three interacting psychological components, rather than a simple linear causal chain. First, at the emotional level, maternal anxiety may trigger threat overamplification and emotional sensitivity dysregulation, causing individuals with maternal anxiety to perceive child distress or routine exploration as significant dangers. Second, at the cognitive level, individuals with maternal anxiety may display attentional bias toward threat cues and engage in catastrophic interpretation of ambiguous situations, while their cognitive reappraisal capacity may become impaired, potentially leading them to justify intrusive interventions as necessary protection. Third, at the behavioral level, overprotective actions (e.g., immediate soothing, activity restriction) produce rapid but temporary anxiety relief. Crucially, this relief functions may as a negative reinforcement signal, increasing the likelihood that overprotection will be repeated in similar future situations. These three levels may continuously feed back into each other: emotional amplification biases cognition, cognitive distortions justify more extreme protection, and behavioral relief reinforces both emotional and cognitive patterns, forming a self-perpetuating psychological closed loop.
At the neural level, the model further attempts to elucidate how maternal anxiety drives overprotective behavior through three key pathways—threat amplification, cognitive dysregulation, and reward reinforcement—thereby achieving cross-level integration. Emotional triggering serves as the starting point of the model. Hyperactivation and heightened vigilance of the amygdala (AMY) correspond to the “emotional response” component of the closed loop, providing the initial driving force for the entire circuit. The connection between the medial prefrontal cortex (mPFC) and the AMY plays a key role in mediating fear and anxiety-like behaviors. Reduced regulatory function of the mPFC and its abnormal connectivity with the AMY lead to threat assessment bias and catastrophic thinking, which correspond to the “cognitive bias” component of the loop. The mPFC fails to effectively “brake”—that is, inhibitory control is weakened—resulting in amplified emotional signals that drive overprotective behavior. This altered neural functional state may lead individuals with maternal anxiety to adopt risk-avoidant decisions (e.g., early intervention or restriction) even in low-threat or uncertain situations—such as a child playing independently in a safe environment—as a preventive coping strategy. Such behaviors may eventually be marked as “effective” by the reward system (the Ventral Tegmental Area (VTA)-Nucleus Accumbens (NAc) dopamine pathway) and become consolidated into habitual actions through negative reinforcement. Thus, in the neural closed loop underlying maternal anxiety-driven overprotective behavior, the VTA-NAc reward pathway serves both as a reward pathway for emotional relief and as a reinforcement mechanism for behavioral consolidation, constituting a key downstream executive node of the loop. The three nodes described above do not operate in a linear cascade but instead form a dynamic interactive network, which may ultimately trap the entire system in a vicious cycle of “anxiety-protection-transient relief-increased vulnerability to anxiety,” making overprotective behavioral patterns increasingly stable and automatic.
The formation of maternal anxiety-driven overprotective behavior does not stem from a single factor but may result from the dynamic coupling of psychological, neural, and social dimensions. Through a systematic review and synthesis of existing literature, this paper proposes an “emotional-cognitive-behavioral closed-loop model”, replacing fragmented factor-based accounts with a dynamic systems perspective. In addition, the model introduces “anxiety relief as reward” via negative reinforcement—a mechanism adapted from addiction and avoidance research—to explain why overprotective behavior becomes habitual and resistant to change, rather than merely attributing it to cognitive errors. Finally, it attempts to achieve cross-level integration by mapping psychological and neural levels onto each other, enabling future studies to test causal pathways rather than mere correlations. In conclusion, this review does not merely summarize existing knowledge but provides a mechanistic, integrative, and actionable theoretical framework that explains the formation, maintenance, and potential impact of maternal anxiety-driven overprotective parenting, thereby offering new directions for both basic research and clinical translation.

Key words: maternal anxiety, overprotection, emotion-cognition-behavior closed loop, neural mechanisms

中图分类号: