ISSN 1671-3710
CN 11-4766/R

›› 2011, Vol. 19 ›› Issue (9): 1347-1354.

• 研究前沿 • Previous Articles     Next Articles

Neuroendocrine Response and Its Mediators Induced by Acute Psychosocial Stress

QI Ming-Ming;ZHANG Qing-Lin;GUAN Li-Li;YANG Juan   

  1. (School of Psychology, Southwest University;, Chongqing 400715, China)
    (Key Laboratory of Cognition and Personality (SWU), Ministry of Education, Chongqing 400715, China)
  • Received:2011-01-21 Revised:1900-01-01 Online:2011-09-15 Published:2011-09-15
  • Contact: YANG Juan

Abstract: Acute psychosocial stress is thought to induce neuroendocrine response by hypothalamus- pituitary-adrenal axis (HPAA) and sympathetic-adrenal medulla axis separately. Salivary cortisol is a stable indicator of HPAA activity response to acute psychosocial stress. This article focuses on studies which have employed the Trier Social Stress Test (TSST) or its modified version for stimulation of HPAA and discusses the role of various mediators (e.g., demographic factors, biological factors, and training) of the acute specificity stress response to psychosocial stress. Authors proposed that neuroendocrine responses to acute psychosocial stress await longitudinal studies.

Key words: acute psychosocial stress, Trier Social Stress Test (TSST), specificity neuroendocrine response, hypothalamus-pituitary-adrenal axis (HPAA)